Thanks in advance... Yeah I saw the film on the smoking tooth some months ago... I don´t have any mercury filling fortunatelly, so the poisoning efect is not may case... I think my case have cervical (I go to chiros 1 a month)and hear loss origin... some people experience hear loss when about 50 years old, and hear loss is directly associated with tinnitus... Tinnitus hv been around for thousands of years but now, with all the enviromental chemical agressions plus ipods and war we are having more and more cases... I copied some info about what the tinnitus community thinks the origins are.. just as matter of ilustration...
Subjective tinnitus is complicated by an inability to obtain an objective measure of its severity, and its cause is unknown. The symptom of tinnitus can be likened to headache with multiple causes. It may be caused by abnormal conditions in the cochlea, the cochlear nerve, the ascending auditory pathway, or the auditory cortex. It has been postulated that the cochlear hair cells injured by noise or head trauma may discharge repetitively, stimulating nerve fibers to discharge synchronously in a way that the central auditory system cannot discriminate from actual sound. In the central nervous system or in the auditory pathways, spontaneous activity within individual auditory nerve fibers may also be synchronized because of injury or metabolic abnormality, resulting in tinnitus. It is also possible that hyperactivity in the nuclei of the ascending auditory pathways may stimulate the auditory cortex in a similar manner. An alternate theory proposes that injury to cochlear integrity from any cause reduces the suppressive influence of the central nervous system, allowing increased neuronal activity higher in the auditory system.
several etiologic factors are prominent. These are classified as otologic, cardiovascular, metabolic,
neurologic, pharmacologic, dental, and psychologic factors. , the largest number of patients with tinnitus appear to have a history of noise exposure or are experiencing presbycusis. In both instances, there is a high-frequency neurosensory hearing loss. Seventy-five percent of patients have a 30+ dB hearing loss from 3 to 8 kHz. This hearing loss is the single most consistent factor in patients with tinnitus.
cardiovascular disease is consistent with the age group (>60 years), but it is still likely that hypertension is a major factor in the onset or severity of the patient's disease.
Thyroid dysfunction can be associated with tinnitus. Hyperthyroidism, by increasing cardiac output, can cause a pulsatile or rushing noise.
Hyperlipidemia (too high blood fat content) is increasingly reported as a factor in tinnitus, particularly in association with fluctuating neurosensory hearing loss and associated dizziness. Vitamin A and/or B deficiency has been described as causing tinnitus.
Whiplash injuries have initiated tinnitus, suggesting that abnormal proprioceptive input from nerve fibers in the neck and shoulder or possibly brain stem
injuries are factors.
Past meningitis (brain infection)may be the cause of tinnitus. Multiple sclerosis can also have severe tinnitus in its constellation of symptoms.
Aspirin and aspirin-containing compounds were identified as the most common inciting medications. As little as 600 to 1000 mm/day of aspirin can create symptoms. Aspirin-containing medications, such as Percodan, Darvon, Bufferin, or Ecotrin, are often overlooked as possible causes of tinnitus.
Nonsteroidal anti-inflammatories, including Naprosyn and ibuprofen, are frequently implicated but not often considered. The effect of these drugs is similar to
aspirin, although not as severe.
Antibiotics, chiefly the aminoglycosides, cause tinnitus. It occurs in most cases of antibiotics used concurrently with diuretics.
Quinine-containing compounds and the synthetic analogues can elevate the severity of tinnitus.
Mercury, arsenic, lead, and other heavy metals in high doses can cause symptoms. A thorough history and alteration of medications are essential components in
treating patients with this complaint.
Temporomandibular joint disorders and dental abnormalities must be considered in taking the general history, in the physical examination, and in devising a
treatment course for tinnitus. Forty-five percent of patients with severe tinnitus describe active temporomandibular joint problems at some time. Thirty-eight percent of patients who have severe tinnitus describe it as concurrent with an increase in the severity of their temporomandibular joint complaints. The
tinnitus is generally low pitched, rough, and associated with a feeling of fullness in the ear.
Psychologic factors play a major role. Stress often increases the perception of tinnitus severity, and depression frequently accentuates the complaint. In some
cases, the tinnitus itself may be the cause of the psychologic disorder. Sixteen percent of the population with tinnitus admitted to depression. Many more
probably were unwilling to admit to it or did not recognize the symptoms. Some studies concluded that 20% to 50% of patients were clinically depressed, and
about half the depressed patients had a long history of depression before the onset of tinnitus.
Sorry to post such a big message, I thought necessary to expand your info... this is real damaging... there a people killing themselves because of this....
Regards a Lv to all....