Re: Up-date on [clonazepam experiment
My opinion, based on tentative collection of data; I am convinced beyond a shadow of a doubt that my parasites--[coccidian protozoa/sporozoa] cyclospora, and cryptosporidium, have a direct and strong association with the CNS and use g-protien receptor pathways. I cannot establish the exact reason, but I believe it has to do with t-cell modulation. It can also do with communication between parastites [no proof, and a bit of a stretch] The
parasites inhibit many glandular functions, at low levels. For example, it prevents the submandibular glands from producing certain histidine products.
After slowly bringing my dosage down, my symptoms are rising at an equal rate.
I believe the drug acts as a blocking agent for isoform neurotransmitters produced by these protozoans. If the dosage was rised to about 6ml at maximum peak, I think it would be enough to cause a massive increase in plasma cells, and clean out most remaining spores and oocyts. But this is a high level, and could be dangerous if not regulated properly. I am also interested in other drugs which may do the job more efficiently and safely, such as a novel peptide supplement.