Here's a link to a case study where a man had Iodine contrast and had "subclinical hyperthyroidism" which eventually lead to hospitalization due to thiamine deficiency. Of course, it took them a while to figure it out.
Here's an excerpt:
It is known that thyroid hormone stimulates oxidative metabolism and ATP utilization which induces the upregulation of the Krebs cycle.12 Thiamine is an important coenzyme for pyruvate dehydrogenase, the enzyme converting pyruvate to acetyl CoA in the mitochondria for use in the Krebs cycle, and it is also required by α-ketoglutarate dehydrogenase which is a Krebs cycle enzyme. Without thiamine, the Krebs cycle is ineffective in carbohydrate metabolism. It has been shown in rats that there is an increase in mitochondrial mass in the hyperthyroid state.13 Therefore, it is postulated that hyperthyroidism is a hypermetabolic state in which there is increased utilization of thiamine. The hypermetabolic state may precipitate Wernicke’s encephalopathy in a patient whose thiamine stores are depleted secondary to prolonged starvation.
You will probably find relief from a B-complex, but it's really important to start with thiamine. OR, you can request a B1 injection. Anytime thiamine deficiency is suspected it should be treated aggressively and with injectable thiamine in the coenzyme form, because it's very hard to recover with oral thiamine when stores of thiamine are low. It puts a strain on the liver which has to convert thiamine into the active form.