I think you are largely in the right area in what you say. I think it is similar to TMAU type II. One way to know if it was a similar issue to TMAU type I (but obviously something different to choline) would be a gene test for FM03 which they do have and use in circumstances where individuals are suspected of having Type I TMAU. If the FMO3 gene (which is instrumental in oxidising and thus neutralising) was compromised in some way it would be evident. This would be helpful as it would indicate whether dietry changes for life are necessary (Type I) or whether it is more of a dysbiosis issue which would (subject to other potential conditions) be potentially curable and not require such a damning prognosis such as complete dietary change for life.