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The link between adrenal fatigue and DNA methylation
 

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Published: 10 years ago
 

The link between adrenal fatigue and DNA methylation


I found this link just a minute ago.


I've been taking Cell Food's liquid SAM-E for about a week now. I recently had a really hard crash. I don't know if it was related to me cutting back the pregnenolone and dhea or taking "Super African Mango 1200". But after a few days of taking the Mango, I developed a weird pain on the top outside of my left foot, a couple of days later, my right knee weakened to the point that it would just buckle and I started waking up nauseous. I recovered with isocort (the pain and weakness disappeared within 30 minutes of taking isocort) and have stopped taking it as symptoms have gone. But this weekend I was as they say now "a hot mess". I was weepy and exhausted. I went back on the preg and the dhea and picked up some licorice and sam-e. I also went back on 25mg of elavil. I've been putting the sam-e in 2 gel caps 'bout 10 drops each and taking 2 caps 2x a day. Today, I took the dog for a walk. I'm tired but not wiped out. I'm going to go grocery shopping in a bit before I have to pick up the kid from school.

Any way, what drove me to start taking Sam-e, was I remember seeing an interview with Cher a few years ago. She had suffered a bout of chronic fatigue and credits Sam-e for giving her life back. So I've been searching on the web for info relating to sam-e and adrenal fatigue and found this article. I haven't had a chance to read it yet, but got excited with this little blurb: It explains so much that is going on with me right now!!!

Anyway, just wanted to get this posted, before my extremely short term memory completely forgets!!!

TTFN!


http://findarticles.com/p/articles/mi_m0ISW/is_262/ai_n13675760/



"It is my clinical experience that as soon as a patient's DHEA-S falls to below 160 the ability to make methyl groups nosedives as well. These patients may then present with symptoms of Depression (inability to synthesize S-adenosylmethionine), joint pain (inability to make methylsulfonylmethionine), and gastric acid reflux disease (inability to make betaine or trimethylglycine), to name a few. Not only does the ability to make methyl groups decrease, but the ability to convert to a methylated product is also compromised. For example, in chronically ill individuals the use of B12--as either the cyanocobalamin or the hydroxocobalamin form seems to do little to improve fatigue or mental functioning. The ideal compound to replenish B12 is methylcobalamin--the only active form. In each case, oral supplementation with the missing methyl-containing substrate ameliorates the symptoms. In each of the scenarios listed, the severity of the illness correlates with the level of the reduced or deficient DHEA-S and the concomitant elevated homocysteine level. The elevated homocysteine level is not only a marker for inflammation, but it is a marker for deficient B vitamins as well. The stage is now set for abnormal DNA methylation and the induction of cancer."
 

 
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