And it keeps coming back to the liver, as you posted Newport. Ugh, I don't like it, but I'm getting the picture. Lymes is one of the things that produces excess ammonia, which needs to be converted in the liver to urea for it to be eliminated through the kidneys. If the liver is sick and not able to convert it, it goes past the liver- to the brain.
L-ornithine-L-aspartate stimulates the urea cycle, and has shown encouraging results in randomized controlled trials.
A couple of other things I found:
Most ammonia in the body forms when protein is broken down by bacteria in the intestines. The liver normally converts ammonia into urea, which is then eliminated in urine.
Ammonia levels in the blood rise when the liver is not able to convert ammonia to urea. This may be caused by cirrhosis or severe hepatitis.
>>>Liver problems are the typical cause of elevated ammonia, which does
lead to encephalopathy.
Learning more from Cutler:
Also known as hyperammonia.
Low manganese on hair test is suggestive but not definitive
Onset or worsening of symptoms with protein or amino acids
Elevated orotate, citrate/isocitrate on urine organic acids test
Ammonia blood test is often not done correctly
Things that make elevated ammonia worse
Citrate, malate and amino acid chelate supplements
Supplements that help with excess ammonia
Alpha-Ketoglutarate (AKG), Arginine Alpha-Ketoglutarate (AAKG)
Ascorbic acid form of Vitamin C with meals
Betaine HCL with meals
Butyric acid / butyrate
Inulin and FructoOligoSaccharides (FOS)
Citrulline with arginine
Protease enzymes to help digest proteins
Lactulose (prescription) is supposed to help