I wouldn't have made the connection with what was causing the noticable reaction to the Amino's had it not been for what Cutler said about the ammonia/glutamine, and then connecting Lyme/ammonia. It was a quick reaction, and happened all 3 times that I experimented...finally ruled out the others and pinpointed it to the Glutamine.
I've known about what Hulda says about the Ornithine/ammonia, and took it while killing the larger parasites- dang, I'm kicking myself for having over-looked it (until now) to go with the Lymes. Important piece of information that shouldn't be over-looked.
>>> reminded me of some symptoms reported by many of my extreme chronic fatigue, fibromyalgia, multiple sclerosis, ALS, and Lyme patients. Subsequent testing revealed that all these people had localized ammonia accumulations primarily in the brain, heart, liver, and gums caused by the bacteria Borrelia burgdorferi (Bb), the causative agent in Lyme disease.
It would seem that Bb releases ammonia, which is converted to glutamine by way of the glutamine synthetase pathways, leading to localized swelling of astrocytes (brain cells). This ammonia-induced glutamine accumulation may cause dysfunction of astrocytes, which leads to impairment of vascular reactivity (other than through a decrease in arginine availability for nitrous oxide) and increased production of nitric oxide (Toshiki 2000). It has been reported that many neurological problems are caused by either congenital or acquired hyper-ammonemia (Albrecht 1998).
All ammonia-related pathological changes predispose these individuals to cerebral allergies, due to alterations in the blood brain barrier, and subsequently allow larger molecules, such as common dietary amino acids, to pass through to the brain. Neurotransmitter receptors and function is impaired, resulting in possibly the most common symptom: altered brain energy metabolism, leading to cognitive brain dysfunctions, i.e., the total fatigue of the mind when forced to read, talk, or think for extended periods.