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Amazing study, this what we need to do? Shocking, isn't it?
demi Views: 3,106
Published: 15 years ago

Amazing study, this what we need to do? Shocking, isn't it?

Check this out. This study proves that eating high cholesterol diet lowers you chlesterol and significatly increases bile production and therefore removes your cholestrol accumulation in Liver. This is simple AMAZING.
Imagine, all of those years those "Doctors" were telling us not to eat cholesterol. There were simple killing us whith their lack of knowledge.

The effects of dietary cholesterol on the regulation of
cholesterol synthesis have been studied intensively since
Schoenheimer and Breusch first observed a relationship
between the two (10, 14). The negative feedback effect,
recently reviewed extensively by Dietschy and Wilson (5), is
achievable only through cholesterol carried to the liver via
the lymph in chylomicron form (15). Moreover, there is
evidence that the effect may also be related to tissue
concentrations of ester cholesterol, as shown in Table 3 and
as suggested earlier (4).
These latter studies, and others (16), suggest that this
feedback mechanism, together with the ability of the liver to
enhance bile acid synthesis, may in a sense be protective in
that it acts to prevent cholesterol accumulation and to
promote maximum removal from the organism. The liver,
under the impact of a high-cholesterol diet, at once greatly
expands its ability to convert hepatic cholesterol to bile acid,
while at the same time almost totally ceasing de novo
synthesis of sterols. The present study underscores this point
by showing that hepatic bile acid formation is intimately
tied to the cholesterol feedback mechanism, through which
a dual, reciprocating precursor supply for bile acid is made
available. There is no quantitatively significant mechanism
by which bile acid synthesis is able to proceed from acetate
when the synthesis of cholesterol is suppressed, although, as
indicated in Table 1, such separate synthesis may occur in
minor amounts. The observation that mevalonate, when
substituted for acetate, can be converted to bile acid in the
liver of the high-cholesterol-fed animals further emphasizes
this close relationship, since 3-hydroxy-3-methylglutaryl
coenzyme A reducĂase,
the critical dietary cholesterol-sensi
tive enzyme (13), is then bypassed.

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