Last year you answered a few questions about treating my mother's lyme disease....she took a course of the herbs and the symptoms cleared up. (as a side note, the two courses of antibiotics didn't have any effect on the symptoms) However, it seems as though once again this year she is having the same symptoms and has started to take the blend again. Once again, the herbs are helping a great deal with the symptoms.
My questions are...is is likely that the issue was never fully resolved and has flared up again? And how long can/should she take the herbal blend to fully correct this issue?
It would be interesting to know what symptoms she is having exactly. The reason is that Lyme disease is actually quite rare, yet largely over diagnosed based on antibody tests and PCR that have no real reliability. But Lyme shares symptoms with various diseases and disorders so it is often used a label like chronic fatigue or fibromyalgia are.
It is is truly Lyme then she could be having symptoms even though the bacteria has been killed off since the bacteria can also components of the immune system and possibly even lead to autoimmunity.
Still it is important that other possibilities are ruled out, especially if she is not likely to have been exposed to deer ticks and never developed the bullseye rash. So le's start specifically with what symptoms she has had and is having now.
There is not test for fibro or chronic fatigue.
They are idiopathic diseases.
Actually these are syndromes, which are not diseases at all. But there is a diagnostic test for fibromyalgia. I have posted on the diagnostics of fibromylagia previously on this forum.
What they are is UNDIAGNOSED LYME (along with Lou Gehrigs, ALS, MS)
(I'm not in the mood to dispute this with anyone who comes in to the thread - so just leave it at that)
I disagree. ALS is Lou Gehrig's to begin with, which has been linked to a viral infection. Same for MS. Lyme is bacterial.
In addition, these autoimmune disorders do not show the characteristic bullseye seen in most actual Lyme patients.
Lyme is actually rare since it takes days of the tick being on the person to create an infection. Most people will find the tick much faster than that.
Actually these are syndromes, which are not diseases at all. But there is a diagnostic test for fibromyalgia. I have posted on the diagnostics of fibromylagia previously on this forum. Yes - they are not diseases. Correct. Waste of time getting ANY test done for Fibro. TREAT THE INFECTIONS, METALS, PARASITES, ETC Viral infections come with Lyme Disease -XMRV, EBV, HHV, etc So I don't disagree that a viral infection is playing a role. But Lyme(borrelia) is at the bottom of it. It's known as the gatekeeper. Once it brings your system down all of the viruses, bacteria, parasites, etc that your body has been harboring or keeping at bay now come out to play. You also then must attend to metals, yeast, endocrine system, and gut.
Let's not forget Lou Gehrig had a summer home in none other than Lyme, Connecticut. He had undiagnosed Lyme disease. In addition, these autoimmune disorders do not show the characteristic bullseye seen in most actual Lyme patients. Lyme is actually rare since it takes days of the tick being on the person to create an infection. Most people will find the tick much faster than that. Utter bullsh!t. You don't need a bullseye rash to be positive for Lyme Disease. I Never had one. And the autoimmune disorder is CAUSED by the bacteria/infection. Again Hvergarth - I hope anyone reading this who may suspect Lyme Disease ignores your comments because they are very inaccurate.
I disagree. ALS is Lou Gehrig's to begin with, which has been linked to a viral infection. Same for MS. Lyme is bacterial.
Actually these are syndromes, which are not diseases at all. But there is a diagnostic test for fibromyalgia. I have posted on the diagnostics of fibromyalgia previously on this forum.
Yes - they are not diseases. Correct. Waste of time getting ANY test done for Fibro. TREAT THE INFECTIONS, METALS, PARASITES, ETC
Once again, there is a diagnostic test for fibromyalgia. But since it can also share symptoms with other conditions it is important to get an accurate diagnosis. Going off half cocked treating for infections, metals, parasites, etc. without even knowing what a person is possibly dealing with is ludicrous.
I disagree. ALS is Lou Gehrig's to begin with, which has been linked to a viral infection. Same for MS. Lyme is bacterial.
Viral infections come with Lyme Disease -XMRV, EBV, HHV, etc
It is true that viruses can infect bacteria. But that does not mean that it is common or happening with the Lyme bacteria. So if you are going to make wild, unfounded claims like this please provide some real evidence from credible sources. Otherwise the claims remain speculation, not fact.
So I don't disagree that a viral infection is playing a role. But Lyme(borrelia) is at the bottom of it.
See my last comment.
It's known as the gatekeeper. Once it brings your system down all of the viruses, bacteria, parasites, etc that your body has been harboring or keeping at bay now come out to play.
More speculation.
You also then must attend to metals, yeast, endocrine system, and gut. Let's not forget Lou Gehrig had a summer home in none other than Lyme, Connecticut.
So what? That is a long stretch to claim that he had Lyme simply because he lived in a town named Lyme!!! What about people who develop ALS in parts of the world where deer ticks are not an issue?
He had undiagnosed Lyme disease.
If it were undiagnosed then how do you know he actually had the bacteria? Again nothing but pure speculation!!!
In addition, these autoimmune disorders do not show the characteristic bullseye seen in most actual Lyme patients.
Lyme is actually rare since it takes days of the tick being on the person to create an infection. Most people will find the tick much faster than that.
Utter bullsh!t. You don't need a bullseye rash to be positive for Lyme Disease.
You need to read more carefully, because that is not what I said. I said the ONLY way to confirm Lyme is to have the characteristic rash. Yes, a small number of people may not develop the rash, but without the rash it is virtually impossible to confirm infection. Why? Simple, because the antibody tests and PCR tests used for "diagnosis" are EXTREMELY inaccurate. Antibody tests in particular are notorious for creating false positives.
In addition, you totally missed my point. The rash appears in the majority of people that actually have the Lyme bacteria. Yet this rash does not appear in MS, ALS or the various other diseases you have tried to link to Lyme. In other words if these people REALLY did have Lyme then they would most likely have developed the characteristic rash, which they do not.
I Never had one.
Good for you. So you may be one of the many misdiagnosed based on the inaccurate antibody and PCR tests.
And the autoimmune disorder is CAUSED by the bacteria/infection.
Not entirely. Microbes are common triggers for autoimmunity, but it is the adrenal dysfunction that actually leads to the overproduction of low affinity antibodies that are the hallmark of autoimmunity.
Again Hvergarth - I hope anyone reading this who may suspect Lyme Disease ignores your comments because they are very inaccurate.
We are going to have to agree to disagree because I see your comments as being the very dangerous ones since they are merely speculation severely lacking any credible evidence. Like assuming Gerhig had undiagnosed Lyme because he lived in a town named Lyme. Using your rationale then people from Blue Ball, Delaware should go to Hooker, Oklahoma then move to Intercourse, Pennsylvania and finally Climax, Colorado.
And I would hate to see what the people in Square Butt, Montana look like!!!
So what? That is a long stretch to claim that he had Lyme simply because he lived in a town named Lyme!!! What about people who develop ALS in parts of the world where deer ticks are not an issue? Lyme is very much prevalent all over the world. Not just caused by deer ticks. Only ignorant Americans think that way.
You need to read more carefully, because that is not what I said. I said the ONLY way to confirm Lyme is to have the characteristic rash. That's non-sense. Blood tests can show you. Funny how that isn't enough PROOF enough for you (because it dosn't fit your agenda) You make up some non-sense about cross reaction. gtfoh Yes, a small number of people may not develop the rash, but without the rash it is virtually impossible to confirm infection. Why? Simple, because the antibody tests and PCR tests used for "diagnosis" are EXTREMELY inaccurate. Antibody tests in particular are notorious for creating false positives. LOL - ok In addition, you totally missed my point. The rash appears in the majority of people that actually have the Lyme bacteria. Yet this rash does not appear in MS, ALS or the various other diseases you have tried to link to Lyme. In other words if these people REALLY did have Lyme then they would most likely have developed the characteristic rash, which they do not. As I referenced to in my other post.... I guess the 100s of patients I've come aross (Uncle included) who were once WRONGLY diagnosed with MS, ALS, CFS only to later test positive via Western Blot or spinal tap for Lyme Disease were just imagining things?
Good for you. So you may be one of the many misdiagnosed based on the inaccurate antibody and PCR tests. LOL You're a stubborn man
We are going to have to agree to disagree because I see your comments as being the very dangerous ones since they are merely speculation severely lacking any credible evidence. Like assuming Gerhig had undiagnosed Lyme because he lived in a town named Lyme. Using your rationale then people from Blue Ball, Delaware should go to Hooker, Oklahoma then move to Intercourse, Pennsylvania and finally Climax, Colorado. As long as you don't delete my post so others can see I'll be fine agreeing to disagree.
And I would hate to see what the people in Square Butt, Montana look like!!!
So what? That is a long stretch to claim that he had Lyme simply because he lived in a town named Lyme!!! What about people who develop ALS in parts of the world where deer ticks are not an issue?
Lyme is very much prevalent all over the world. Not just caused by deer ticks. Only ignorant Americans think that way.
Again you lack any credible evidence to support your claim. And the few links you did provide were full of assumptions and contradictions.
You need to read more carefully, because that is not what I said. I said the ONLY way to confirm Lyme is to have the characteristic rash.
That's non-sense. Blood tests can show you. Funny how that isn't enough PROOF enough for you (because it dosn't fit your agenda)
You make up some non-sense about cross reaction. gtfoh
LOL!!! Great example of projection. Apparently for someone who works with Lyme patients I would think you would know about how antibody tests work, including their shortcomings. I guess you are determined that I continue proving how little you know about the subject to the point where you have zero credibility. Well if that is what you want here it goes. Information on the "cross reaction", which as I pointed out previously is a well known problem really called serological cross reactivity:
http://www.everythingbio.com/glos/definition.php?word=cross-reactivity
"Definition of cross-reactivity :
The ability of an antibody to react with or bind an antigen that did not stimulate its production. This is sometimes seen in western blots, where an antibody detects other proteins other than the one the antibody is specific for. "
http://en.wikipedia.org/wiki/Cross-reactivity
"Cross-reactivity is the reaction between an antibody and an antigen that differs from the immunogen. It is sometimes also referred to as crossimmunity or cross-protective immunity.[1] A few examples of cross-reactivity have been confirmed in humans, one of which involves influenza virus-specific CD8+ T cell and hepatitis C virus antigens.[2]
An adaptive immune response is specific to the antigen that stimulated it (called the immunogen). However, many naturally occurring 'antigens' are a mixture of macromolecules (e.g. from pathogens, toxins, proteins, pollen) comprising several epitopes. Contact with a complex antigen such as a virus will stimulate multiple immune responses to the virus' different macromolecules as well as the individual epitopes of each macromolecule. For example, the tetanus toxin is a single protein macromolecular antigen but will stimulate many immune responses due to the tertiary structure of the protein yielding many different epitopes."
And guess what, even Lyme can cross react!!!:
http://www.lymeneteurope.org/forum/viewtopic.php?f=5&t=1686
J Infect Dis. 1987 Jul;156(1):183-8.Links
"
Cross-reactivity in serological tests for Lyme disease and other spirochetal infections.
Magnarelli LA, Anderson JF, Johnson RC.
Serum specimens from 163 persons with Lyme disease, tick-borne or louse-borne relapsing fever, yaws, syphilis, leptospirosis, or Rocky Mountain spotted fever were analyzed to assess the specificity of indirect fluorescent antibody (IFA) tests, an enzyme-linked immunosorbent assay (ELISA), and microscopic agglutination (MA) procedures.
Strong cross-reactivity occurred when sera from individuals with Lyme disease, tick-borne relapsing fever, and louse-borne relapsing fever were tested against heterologous Borrelia antigens. Antibodies to Borrelia burgdorferi bound to Treponema pallidum in immunofluorescence tests for syphilis.
Sera from subjects with syphilis cross-reacted in IFA tests and the ELISA for Lyme disease. Immunoglobulin antibodies to Borrelia or Treponema spirochetes, however, did not react with serovars of Leptospira interrogans in MA or IFA tests, and the prevalence of false-positive results in the reciprocal analyses was negligible."
"B. burgdorferi cross-reactions:
kDa
18: C. pneumonia
23-25: B. Hermsi, leptospirosis (band 25), Yersina, C. pneumonia (band 25)
35: Yersina, C. pneumonia
39: B. Hermsi
41: S. pallidum, L. interrogans, Yesirna, potentially all spirochetes
60: S. pallidum, E. coli, Bartonella, Staphylococcus, M tuberculosis, E. coli
70: HGE
Proteins that could be close enough possibly to get confused, if test or interpretation is sloppy:
kDa
18: S. pallidum (17 kDa)
30: C. pneumonia (29 kDa)
31: L. interrogans (32 kDa)
34: B. Hermsi (35 kDa)
41: C. pneumonia (40 kDa)
45: T. pallidum (47 kDa),
66: M. tuberculosis, E. coli (65 kDa)"
http://www.igenex.com/labtest.htm
"
Table 1 presents the types of tests that are most commonly available for Lyme disease. To provide adequate support for the clinical evaluation, multiple tests should be used. Not only is a correct diagnosis advantageous for the patient, but also ultimately is the most cost effective.
B. burgdorferi spirochetes are affixed to glass slides and usually a fluorescent-conjugated goat antihuman immunoglobulin of either IgM or IgG specificity is used (20). Tests for Lyme disease using IFA have received mixed reviews and some authors believe that the interpretations of IFA assays are overly subjective and that the tests are either functionally insensitive for Lyme-specific antibodies or display considerable cross-reactions with antibodies to other spirochetal organisms (21,22). Magnarelli et al (23,24) and Mitchell et al (20) supported IFA if used in conjunction with a clinical evaluation. Mitchell's study with the IgM IFA showed excellent specificity and no observed cross-reactivity with infectious mononucleosis (n = 20), rheumatoid arthritis (n = 19), systemic lupus (n = 22), syphilis (n = 13), streptococcal sequelae (n = 20) or healthy subjects. Mitchell related the success of this test to the quality of the substrate slides and the level of experience of the technologists, and concludes that IFA microscopy becomes less subjective with experience.
ELISA for B. burgdorferi has been available since 1984 (25). Most commercial assays use a whole cell sonicate of B. burgdorferi. Complete descriptions of methods for a Lyme ELISA can be found in the publications by Craft et al (25), Magnarelli et al (23), and Russell et al (21). Standard ELISA techniques have been employed (26) in all these assays.
There are a large number of commercial ELISA tests available. A review of past proficiency events by CAP and the NYS Health Department show the relationship between the various tests. Most commercial ELISA tests have comparable sensitivity and specificity because they were made to compare to one another for the FDA 510K process. However, most are inadequate as a screening test because they were not designed by the manufacturers to be sensitive at the 95% level, which is required for screening (14). A substantial change in the 510K approval process would be required to make the ELISAs for Lyme disease diagnosis more sensitive.
The goal for a new generation of ELISAs should be sensitivity for the more unique and specific B. burgdorferi antigens that are visualized in the Western blot (Figure 1). They are Osp A (31 kDa), Osp B (34 kDa), Osp C (23-25 kDa), 39 kDa, and 93 kDa (27-32). Initially, some investigators identified 93 kDa as 94 kDa and Osp C as 22 kDa. While most ELISAs do have reactivity to these antigens, because they are prepared with a sonicate of B. burgdorferi, they also have reactivity against 41 kDa, 58 kDa, 66 kDa, and 73 kDa. While the later antigens are components of B. burgdorferi, they also have considerable cross-reactivity to other spirochetes, heat-shock proteins, and some viruses (33)."
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC267918/
Cross-reactivity of nonspecific treponemal antibody in serologic tests for Lyme disease.
http://www.ncbi.nlm.nih.gov/pubmed/18844689
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1287758/
Evaluation of a Western Blot Method for the Detection ofYersinia Antibodies: Evidence of Serological Cross-Reactivity between Yersinia Outer Membrane Proteins andBorrelia burgdorferi
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1287758/table/t3/
Cross-reactivity observed for the Yersinia CF assay, Western blot assays, and ELISAs with samples positive for antibodies to various organisms
So now that you have thoroughly embarrassed yourself by showing your complete lack of knowledge of the subject you still want to keep this up?
Yes, a small number of people may not develop the rash, but without the rash it is virtually impossible to confirm infection. Why? Simple, because the antibody tests and PCR tests used for "diagnosis" are EXTREMELY inaccurate. Antibody tests in particular are notorious for creating false positives.
LOL - ok
You should not have laughed so much before doing your homework since you have just been COMPLETELY discredited!!!
In addition, you totally missed my point. The rash appears in the majority of people that actually have the Lyme bacteria. Yet this rash does not appear in MS, ALS or the various other diseases you have tried to link to Lyme. In other words if these people REALLY did have Lyme then they would most likely have developed the characteristic rash, which they do not.
As I referenced to in my other post.... I guess the 100s of patients I've come aross (Uncle included) who were once WRONGLY diagnosed with MS, ALS, CFS only to later test positive via Western Blot or spinal tap for Lyme Disease were just imagining things?
And once again the lab tests for Lyme ARE NOT accurate as the evidence I have posted shows. And again, if you were such an expert on the subject then you should have been aware of that. So you have just shown that you are in no way an expert on the subject.
Good for you. So you may be one of the many misdiagnosed based on the inaccurate antibody and PCR tests.
LOL You're a stubborn man
I am not the one making unfounded claims that keep getting disproven.
We are going to have to agree to disagree because I see your comments as being the very dangerous ones since they are merely speculation severely lacking any credible evidence. Like assuming Gerhig had undiagnosed Lyme because he lived in a town named Lyme. Using your rationale then people from Blue Ball, Delaware should go to Hooker, Oklahoma then move to Intercourse, Pennsylvania and finally Climax, Colorado.
And I would hate to see what the people in Square Butt, Montana look like!!!
As long as you don't delete my post so others can see I'll be fine agreeing to disagree.
No, I am not going to delete the posts since they clearly show your lack of knowledge on the subject, and people need to be aware of this. But I am putting a stop to this nonsense by blocking you from this board since you seem to only want to argue over things you clearly have no understanding of. And I am not going to waste any more time addressing your unproven and disproven claims. You can take this garbage over to the Lyme board.
Anaplasmosis is carried by the same ticks that carry Lyme's bacteria, but her symptoms may simply be a repeat of the anaplasmosis.
Titers really mean nothing. These are antibody tests, which are pretty much worthless. So without a specific symptom such as the bullseye rash it is pretty much impossible to accurately diagnose Lyme's disease.
Lyme Disease is under-diagnosed.
Wrong, it is way over diagnosed.
It's an epidemic and soon this country will be forced to wake up. Millions are affected and don't even know it.
That is an impossible claim to prove, so I will not even ask you to try. But Lyme's is actually rare due to the fact that it takes days of the tick being on the person to cause infection. But people will generally find ticks on themselves long before that, not to mention that there are plenty of parts of the country where ticks are not an issue. And even in the areas that they are what percentage of the population is going hiking in the woods to be exposed? It is not transmitted person to person, or through the air, water or food.
It becomes even more complicated because there are plenty of false-negatives (I tested negative twice before testing positive) So people they don't have it - when in fact it's the root cause of all their problems.
False positives are way more prevalent with antibody testing than are false negatives. The biggest problems with these tests are false positives from serological cross reactivity and antibody responses from exposure without chronic infection. For example, if not enough of the bacteria is introduced to cause chronic infection or antibiotics kill off the bacteria there will still be an antibody response that will yield false positives. This is why without the characteristic bullseye rash there is no way to positively confirm Lyme's infection.
Wrong, it is way over diagnosed. *shakes head* Sorry Hvergarth - I love your work but you're out of the loop on this one. That is an impossible claim to prove, so I will not even ask you to try. But Lyme's is actually rare due to the fact that it takes days of the tick being on the person to cause infection. But people will generally find ticks on themselves long before that, not to mention that there are plenty of parts of the country where ticks are not an issue. And even in the areas that they are what percentage of the population is going hiking in the woods to be exposed? It is not transmitted person to person, or through the air, water or food. The fact you call it "Lyme's" tells me all I need to know. It doesn't take days and tick bite isn't the only way it can be passed. My mother passed it to me and my 2 brothers at birth (through placenta). We all have Lyme and I showed my earliest symptoms of it when I was a child. Spirochetes have been found in sperm as well and much research has shown it can be passed sexually (much like Syphillis which is a spirochete infection) False positives are way more prevalent with antibody testing than are false negatives. The biggest problems with these tests are false positives from serological cross reactivity and antibody responses from exposure without chronic infection. For example, if not enough of the bacteria is introduced to cause chronic infection or antibiotics kill off the bacteria there will still be an antibody response that will yield false positives. This is why without the characteristic bullseye rash there is no way to positively confirm Lyme's infection. Complete BS - I hope people reading this take your remarks with a grain of salt. I have first hand experience with this. you apparenly don't. You're beyond knowledgeable and I've appreciated much of the work you provide to this forum. But your knowledge of Lyme Disease is so off base.
Wrong, it is way over diagnosed.
*shakes head* Sorry Hvergarth - I love your work but you're out of the loop on this one.
Not even close.
That is an impossible claim to prove, so I will not even ask you to try. But Lyme's is actually rare due to the fact that it takes days of the tick being on the person to cause infection. But people will generally find ticks on themselves long before that, not to mention that there are plenty of parts of the country where ticks are not an issue. And even in the areas that they are what percentage of the population is going hiking in the woods to be exposed? It is not transmitted person to person, or through the air, water or food.
The fact you call it "Lyme's" tells me all I need to know.
If you prefer Lyme I could use that. But if you want to be nitpicky do you know why the "L" is capitalized? If you know then you should also know that "Lyme's" is acceptable.
It doesn't take days and tick bite isn't the only way it can be passed. My mother passed it to me and my 2 brothers at birth (through placenta). We all have Lyme and I showed my earliest symptoms of it when I was a child.
Show me proof that it can be passed that way. Being that there are no definitive lab tests that can prove the presence that is going to be pretty difficult. But I will be waiting for your evidence.
Spirochetes have been found in sperm as well and much research has shown it can be passed sexually (much like Syphillis which is a spirochete infection)
Again, let's see some evidence that Lyme can be passed sexually and passed through the placenta from reliable sources.
False positives are way more prevalent with antibody testing than are false negatives. The biggest problems with these tests are false positives from serological cross reactivity and antibody responses from exposure without chronic infection. For example, if not enough of the bacteria is introduced to cause chronic infection or antibiotics kill off the bacteria there will still be an antibody response that will yield false positives. This is why without the characteristic bullseye rash there is no way to positively confirm Lyme's infection.
Complete BS - I hope people reading this take your remarks with a grain of salt. I have first hand experience with this. you apparenly don't.
I know more about those tests than you could ever hope to know. If you have "first hand" experience then you would have to be a lab tech or similar, which means that you would have enough knowledge to understand all the various scientific reports explaining how antibody tests and PCR (polymerase chain reaction) are unable to prove the presence of any pathogen. Look them up I have found plenty.
You're beyond knowledgeable and I've appreciated much of the work you provide to this forum. But your knowledge of Lyme Disease is so off base.
Show me proof that it can be passed that way. Being that there are no definitive lab tests that can prove the presence that is going to be pretty difficult. But I will be waiting for your evidence.
James, you don't always need a lab test to know something exists.
I work with Lyme patients on a day to day basis. I can't tell you how many mother's have unfortunately passed borrelia onto their children. (confirmed through blood results and devastating symptoms)
Lyme is arguably the most controversial disease in America today. And just because the CDC ignores it or you haven't read some "evidence" - doesn't mean the truth doesn't exist.
You will find out in due time. In the meantime though...
Here is a link showing Lyme passed during pregnancy (read entire document if you have time)
http://sites.google.com/site/drjoneskids/pregnancy-and-lyme/pregnancy-outcomes
Here is a link showing spirochetes found in semen
http://www.anapsid.org/lyme/bach.html
Here is a link showing you the disconnect between these idopathic illnesses and Lyme
http://floridadetox.com/lyme-disease-found-in-100-percent-of-150-als-patients
Show me proof that it can be passed that way. Â Being that there are no definitive lab tests that can prove the presence that is going to be pretty difficult. Â But I will be waiting for your evidence.
James, you don't always need a lab test to know something exists.
There is a big difference between assuming something and proving something. Look at all the people over on the "Liver Flush Support" forum claiming they are passing gallstones up to the size of baseballs, which is simply impossible. And like you they swear up and down that they are right and that anyone challenging their claims are simply wrong. Yet when you read posts from people that actually sent those big blobs to the lab for analysis it has been confirmed over and over that the blobs were soap stones. So sorry, but I am not going to take someone's opinion as evidence.
I work with Lyme patients on a day to day basis. I can't tell you how many mother's have unfortunately passed borrelia onto their children. (confirmed through blood results and devastating symptoms)
Lyme is arguably the most controversial disease in America today. And just because the CDC ignores it or you haven't read some "evidence" - doesn't mean the truth doesn't exist.
Just because you claim something this does not make it true.
You will find out in due time. In the meantime though...
Here is a link showing Lyme passed during pregnancy (read entire document if you have time)
http://sites.google.com/site/drjoneskids/pregnancy-and-lyme/pregnancy-outcomes
Lot's of evidence against your claim. Did you read these quotes from your own link:?
"Here we can only speculate that the Lyme might have been responsible for the birth defects, as these same types of problems can occur in non-Lyme situations."
"Williams and colleagues conducted a study in a Lyme-endemic area in New York of umbilical cord blood. Of 255 infants tested, 10.2% had detectable antibody to the Lyme spirochete. Of 166 infants born in a non-endemic area, 2.4% had detectable antibodies. The rate of birth defects did not differ significantly between the two groups; however, the first group tended to be of lower birth weight and smaller for their gestational age, and tended to have more jaundice. The authors concluded that these differences were not significantly different. "
"The conclusion was that healthy women who test positive for Lyme are at no increased risk for miscarriage."
"In 1988, Carlomango studied 49 women who had either a 1st or 2nd trimester spontaneous abortion. Six (6) of them (12.2%) tested positive, compared to 3 of 49 women who delivered at term. The authors concluded that there was no statistical significance between the two groups."
And so on and so forth. Note the pattern of NO definitive evidence linking actual Lyme infection to any of the disorders. And when they do attempt to make the link they state the babies are either born dead or die shortly afterward. So how did you and your two brothers survive based on the claims about Lyme causing so much damage to the fetus? In fact, if you actually read your own link you would have see that it is full of speculations and contradictions.
Here is a link showing spirochetes found in semen
http://www.anapsid.org/lyme/bach.html
Again, you need to actually read the links you are going to post. This is from your second link:
" Whether or not further clinical studies with a larger statistical group will support the hypothesis of sexua| transmission remains to be seen."
If you know what a hypothesis is then you would know why the link was worthless as evidence.
Here is a link showing you the disconnect between these idopathic illnesses and Lyme
http://floridadetox.com/lyme-disease-found-in-100-percent-of-150-als-patients
This is a sales site, not real evidence of anything.
And again, if you actually read your own link you would have seen all the contradictions. For example, how one researcher only found an 80% positive test rate for Lyme in ALS patients (keep in mind though the high false positive rate with antibody testing). Then there is another researcher who claims a 100% positive rate for lyme in 150 ALS patients. Yet they mention the other researcher that found ZERO traces of Lyme in all ALS patients tested. But here is the real kicker:
"European University Medical Center studies have proven the Black Mold Trichothecene toxin causes brain cancer, liver cancer, gastric cancer, Multiple Sclerosis, ALS, Parkinson’s, destruction of Chondroblasts [cells that manufacture cartilage], gastrointestinal lining cells and interferes with RNA-DNA replication in all cells."
So wait, here you have been claiming all along that Lyme bacteria was responsible for MS and ALS, but here according to your own link they say that MS and ALS are caused from a black mold, not Lyme bacteria!!!
Hveragherthi:
Unless you have studied and researched Lyme Disease for 10 years in depth and from the leading Lyme Disease medical doctors in the United States and abroad and know how to spell this disease correctly, it is LYME DISEASE, not Lyme's, you honestly have no business what-so-ever spreading false information like this. It is clear from reading your posts that you don't have much of any solid knowledge on this complex disease. After studying it in depth for this long, even I am still learning new things because the medical research continues to build, progress, and expand upon previous work from researchers, doctors, and scientists.
If you want proof, by all means, spend years studying it like others have and spot harassing people who know what they're talking about. Do you own intensive homework and you will find the right answers.
I am done arguing about Lyme disease. Misleading and unproven claims were made by someone that did not have a clue what they were talking about and I addressed it. Even the skimpy evidence that the poster did present showed contradictory evidence to his claims. Then the final straw was when he claimed that cross reactivity was a made up term even though it is well known. I learned about it over 20 years ago.
This site is not about making wild unfounded claims like all autoimmune disorders are from Lyme, or that Lou Gehrig had undiagnosed Lyme because he lived in Lyme, Connecticut. So if people have real evidence to the contrary then present it. But don't come here to insult me or to keep telling me I am wrong when you cannot provide any real evidence to the contrary. I have had more than enough of that crap and again I am not going to keep arguing over this since no contrary evidence has yet to be presented so arguing over people simply saying I am wring without any evidence is just a major waste of time.