Well the argument is far from settled with the so called dissidents. Their questions still remain unanswered.
Now apart from exceptions people who test HIV antibody positve develop AIDS I accept this and do not dispute it, but correlation does not mean causation. My problem of accepting the HIV = AIDS hypothesis is there is AIDS without HIV* and at THE VERY MOST HIV only infects 1 out of every 500 immune cells.
Now it is 24+ years later since Gallo and the U.S goverment declared 'HIV is the probable cause of AIDS' on April 23rd 1984 (the same day that Gallo and the NIH patent HIV antibody tests as evidence of AIDS) and this is where we are:
• No curative medications
• Treatment consists of chemotherapy
• NIH only funds research linking HIV/AIDS
• Research failures are used for fund raising
It is strange that numerous virus vaccines have been developed since Ed Jenner’s pox vaccine in 1793 yet HIV is somehow different. THE ALARM BELLS SHOULD BE GOING OFF BY NOW AS VACCINE RESEARCH SOLELY TARGETING HIV HAS CLEARLY NOT WORKED. THEY HAVE BEEN TRYING TO MAKE A VACCINE NOW FOR A QUARTER OF A CENTURY! AS EINSTEIN SAID THE DEFINITION OF INSANITY IS DOING THE SAME THING OVER AND OVER AND EXPECTING DIFFERENT RESULTS. MAYBE THEY SHOULD OF STARTED BY ISOLATING THE VIRUS IN THE FIRST PLACE THAT WOULD OF BEEN A GOOD START. IS HISTORY GOING TO LOOK AT THIS LIKE THE LYSENKO AFFAIR?
*The CDC even came up with a classification for non HIV AIDS it is called 'Idiopathic CD4 lymphocytopenia(ICL)'.
In 1993 Duesberg collected references for 4,621 HIV-free AIDS cases
from the literature:
Duesberg, P. The HIV gap in national
AIDS statistics. Biotechnology, 11: 955-
The AIDS orthodoxy states that HIV is/can be the SOLE cause of AIDS without CONCLUSIVE peer reviewed experiments, articles or papers this is akin to the university of Liverpools research that found that 97% of women with Breast Cancer test positive for cytomegalovirus(CMT) and then stating CMT causes breast cancer.
Here are some more interesting quotes regarding HHV-6A.
A case of idiopathic CD4 T-lymphocytopenia (ICL) and CMV disease(notice the words 'She had persistently high HHV-6').
"HHV-6A was shown to dramatically accelerate progression from HIV to full blown AIDS in macaques, by causing an early depletion in both CD4+ and CD8+ cells. (Lusso 2007) HHV-6A may cause more of the destruction of lymphoid tissue and apoptosis of immune cells than HIV. Bob Gallo spoke on this subject at the last HHV-6 conference in Barcelona, telling the group that HHV-6A is an important progression factor in AIDS and has been wrongly ignored.
Of note, HIV can not invade CD8 cells until HHV-6A has first infected the cell to induce the CD4+ receptor. (Lusso 1991) HHV-6A induces de novo expression of CD4 messenger RNA and protein in normal mature CD8+ T lymphocytes, rendering them susceptible to infection with HIV".
"Carrigan and Knox, meanwhile, were pursuing a theory that had been considered, and rejected, by researchers earlier - that HHV-6 might be a "co-factor" in AIDS, aiding and abetting HIV in the destruction of victims' immune functions. Soon, Regush relates, Knox was wondering, "was HIV doing any killing, or was HHV-6 the lone assassin?" But in 1997, after Carrigan and Knox found evidence suggesting HHV-6 might be killing alone, the British medical journal The Lancet rejected their paper for publication." --Mark Nichols