You use parasites killing tincture + zapper to kill heartworms.
You can also use Beck Electrification device instead of zapper (silver pulser)
Most likely, heart worms will not be found inside your heart, they are more likely inside your lungs.
Sometimes, heart worms endup other places like eyes, testicles, ovaries, ...
Evolution does not know for boundaries!
Instead of migrating to the heart, the larvae most commonly migrate to the lungs in humans. There the larvae can block vessels causing an infarction. At the site of the infarction, a nodule develops which can be seen on radiographs. Usually the person has few, if any signs of infection. Usual medical treatment is surgical removal of the nodule.
Heartworms have even been found in horses.
If you choose to use herbs, once worms are dead ... your blood will desintegrate their bodies and your liver will convert residues into bile and it will be flushed out.
Humans are an "aberrant" host for heartworms. The microfilaria (baby heartworms) are transferred to humans by mosquitos and they try to follow their normal lifecycle but can't, because they are in the wrong host. This does not prevent them from succeeding in making it partway through their lifecycle, though. So instead of winding up in the heart and pulmonary arteries and living several years, as occurs in dogs, the heartworms usually end up as cysts in the lungs. These cysts look pretty much like lung cancer on an X-ray and a number of humans have had surgery to remove the cyst and/or lung lobes as the result of the resemblance.
In addition, on rare occasions, heartworms find another spot in the body with conditions that support their development and live there for some time. In humans, a spot that has these conditions is the interior of the eye.
So heartworms are occasionally found inside the eye in humans.
You could go to the Pub Med web site: http://www.ncbi.nlm.nih.gov/entrez
and search on the term "dirofilaria immitis human" and among the first few hits will be an article with this title,
"Case report: Unusual location of Dirofilaria immitis in a 28-year old man necessitates orchiectomy".
An orchiectomy is the removal of a testicle.
So treating your dog and cat against parasites is not only good
for their health, it helps to reduce the incidence of a disease that can have serious impact, although rarely, on humans.
By the way, how do you know that you have heartworms?
There are several blood tests used to detect heartworm infection. In the 1960’s, before more sophisticated tests were available, heartworm testing involved looking for the microfilariae in a drop of blood on a microscope slide.
Dark microscope would be the best to use.
A better test, the Knott’s test, was developed to concentrate the microfilariae from a larger portion of blood through centrifuging it. This gave veterinarians a better chance of finding the microfilariae.
Later, filter tests became available. In these tests, the blood cells in the blood were lysed (broken) by a special agent that did not affect the microfilariae. The resulting liquid was then put through a very fine filter. The microfilariae were concentrated on the filter. The filter was stained and examined under the microscope for microfilariae.
Veterinarians soon recognized that some animals could have heartworm infections without having microfilariae in the blood. This occurs if only male worms are present or if the females are not laying microfilariae at the time of the test. It was obvious that better tests were needed.
Serologic tests were developed to identify antigens (small protein and carbohydrate components) of heartworms in the blood stream. There are different varieties of this test. Some test kits run one sample at a time and can be done right in your veterinarian’s office. Others are designed to test multiple samples in large batches. This batch-type of test is generally performed by outside laboratories to which your veterinarian sends your dog’s blood.
Although the antigen tests were much better than the filter test, we still could not identify all cases of heartworm infection because antigen tests will only be positive if female worms are present, since the antigen detected is from the worm’s uterus. If the heartworms were not fully mature, or there were only male worms present, the antigen test result in infected animals would be falsely negative. This means the test result is negative when the animal is really infected.
Serologic tests have now been developed to detect antibodies (proteins to fight off the "invaders") the animal makes against heartworms. This is the test most commonly used in cats. This test will be positive even if only one male worm is present. But this test has a downfall, too. Although it is very good at giving positive results when an infection is present, false positive tests are more common with this test than the antigen test. A false positive result means the test result is positive when no infection is present. One in every 200 antibody heartworm tests in cats may be falsely positive.
There have been rare instances of heartworm infection in people. As of 1995, 97 cases of human heartworm disease have been reported in the United States. Except for Texas and Louisiana, all cases have occurred east of the Mississippi River.
All experts agree that real number of people infected can be up to 100 or even 1000 times larger, cause heartworm would be the last your doctor would suspect.
Heartworm is an occasional parasite of humans. The parasite is usually found in the lung, less often in the heart. The worms form a "coin lesion" in the lung which may be confused with other diseases on x-ray, such as carcinoma. During the last twenty years about 80 cases of human pulminary dirofilariasis have been reported in Florida alone.
Heartworm disease (dirofilariasis) is a serious and potentially fatal disease in dogs. It is caused by a worm called Dirofilaria immitis.
Heartworms are found in the heart and large adjacent vessels of infected dogs. The female worm is 6 to 14 inches (2.3 to 5.5 cm) long and 1/8 inch (5 mm) wide; the male is about half the size of the female. One dog may have as many as 300 worms.
How do heartworms get into the heart?
Adult heartworms live in the heart and pulmonary arteries of infected dogs. They have been found in other areas of the body, but this is unusual. They survive up to 5 years and, during this time, the female produces millions of young (microfilaria). These microfilaria live in the bloodstream, mainly in the small blood vessels. The immature heartworms cannot complete the entire life cycle in the dog; the mosquito is required for some stages of the heartworm life cycle. The microfilaria are therefore not infective (cannot grow to adulthood) in the dog, although they do cause problems.
As many as 30 species of mosquitoes can transmit heartworms. The female mosquito bites the infected dog and ingests the microfilariae during a blood meal. The microfilariae develop further for 10 to 30 days in the mosquito and then enter the mouth parts of the mosquito. The microfilariae are now called infective larvae because at this stage of development, they will grow to adulthood when they enter a dog. The mosquito bites the dog where the haircoat is thinnest. However, having long hair does not prevent a dog from getting heartworms.
When fully developed, the infective larvae enter the bloodstream and move to the heart and adjacent vessels, where they grow to maturity in 2 to 3 months and start reproducing, thereby completing the full life cycle.
Where are heartworms found?
Canine heartworm disease occurs all over the world. In the United States, it was once limited to the south and southeast regions. However, the disease is spreading and is now found in most regions of the United States and Canada, particularly where mosquitoes are prevalent.
How do dogs get infected with them?
The disease is not spread directly from dog to dog. An intermediate host, the mosquito, is required for transmission. Spread of the disease therefore coincides with the mosquito season. The number of dogs infected and the length of the mosquito season are directly correlated with the incidence of heartworm disease in any given area.
It takes a number of years before dogs show outward signs of infection. Consequently, the disease is diagnosed mostly in 4 to 8 year old dogs. The disease is seldom diagnosed in a dog under 1 year of age because the young worms (larvae) take up to 7 months to mature following establishment of infection in a dog.
What do heartworms do to the dog?
Adult worms: Adult worms cause disease by clogging the heart and major blood vessels leading from the heart. They interfere with the valve action in the heart. By clogging the main blood vessels, the blood supply to other organs of the body is reduced, particularly the lungs, liver and kidneys, leading to malfunction of these organs.
Most dogs infected with heartworms do not show any signs of disease for as long as two years. Unfortunately, by the time signs are seen, the disease is well advanced. The signs of heartworm disease depend on the number of adult worms present, the location of the worms, the length of time the worms have been present, and the degree of damage to the heart, lungs, liver, and kidneys from the adult worms and the microfilariae.
The most obvious signs are: a soft, dry, chronic cough, shortness of breath, weakness, nervousness, listlessness, and loss of stamina. All of these signs are most noticeable following exercise, when some dogs may even faint.
Listening to the chest with a stethoscope will often reveal abnormal lung and heart sounds. In advanced cases, congestive heart failure may be apparent and the abdomen and legs will swell from fluid accumulation. There may also be evidence of weight loss, poor condition, and anemia.
Severely infected dogs may die suddenly during exercise or excitement.
American Heartworm Society. Recommended Procedures for the Diagnosis, Prevention, and Management of Heartworm (Dirofilaria immitis) Infection in Dogs. Batavia IL;1995.
American Heartworm Society. Recommended Procedures for the Diagnosis, Prevention, and Management of Heartworm (Dirofilaria immitis) Infection in Cats. Batavia IL;1995.
Calvert, CA. Heartworm Disease. In Birchard, SJ; Sherding, RG (eds): Saunders Manual of Small Animal Practice. WB Saunders Co., Philadelphia PA; 1994;487-493.
Griffiths, HJ. A Handbook of Veterinary Parasitology. University of Minnesota Press, Minneapolis MN; 1978;60-62.
Hendrix, cm. Diagnostic Veterinary Parasitology. Mosby, Inc., St. Louis MO; 1998;126-128, 265-272, 296-298.
Lok, JB; Knight, DH. A review of the treatment options for heartworm infections. Supplement to Veterinary Medicine, June 1997:15-25.
Lok,JB;Knight,DH. Heartworm infections: Strategic timing of testing and prevention. Supplement to Veterinary Medicine, June 1997:4-14.
Losonsky, JM. In Thrall, DE (ed): Textbook of Veterinary Diagnostic Radiology. WB Saunders CO., Philadelphia PA; 1994; 323-325.
Sisson,D. A definitive approach to heartworm disease in cats. Veterinary Forum, April 1998:40-48.
Sousby, EJL. Helminths, arthropods and protozoa of domesticated animals. Lea & Febiger, Philadelphia PA; 1982;307-312.
Wright, JC; Hendrix, CM; Brown. Dirofilariasis. In: Zoonoses updates from the Journal of the American Veterinary Medical Association. American Veterinary Medical Association, Schaumburg IL; 1995:56-61.
Case report: Unusual location of Dirofilaria immitis in a 28-year-old man necessitates orchiectomy.
Theis JH, Gilson A, Simon GE, Bradshaw B, Clark D.
Department of Medical Microbiology, School of Medicine, University of California, Davis 95616-8645, USA.
The fourth case of zoonotic Dirofilaria immitis infection in a California resident is reported. This case was unique in that it involved the spermatic cord, produced significant symptoms, and upon physical examination was suspected of being an incarcerated inguinal hernia.
At surgery frozen sections were inconclusive regarding neoplasia and cord involvement precluded removal of the mass without orchiectomy, which was done. Histopathology revealed a well-preserved immature male D. immitis, the canine heartworm, in a branch of the spermatic artery. The morphological identification of the worm was confirmed by polymerase chain reaction (PCR) studies.
This case illustrates that D. immitis infections can complicate differential diagnosis of extrapulmonary lesions and suggests that the public health significance of this vector-borne filarial worm needs to be evaluated through appropriate epidemiological studies in enzootic areas.
A 44-year-old previously healthy woman presented to her primary
care physician with a 4-week history of dry cough. She had a
27-pack-year smoking history. There was no history of shortnessof
breath, chest pain, hemoptysis, fever, night sweats or weightloss.
She had travelled to Mexico, South Carolina and the Dominican
Republic in the 2 years before the current illness. Physical
examination and routine laboratory investigations, includingcomplete
and differential blood counts, were normal. A chestradiograph
revealed a nodular opacity within the anterior aspectof the right
upper lobe of the lung (Fig. 1, arrow).
Figure 1. Photo: Images courtesy of the
Departments of Pathology and Molecular Medicine (HRLMP) and of
Diagnostic Imaging, St. Joseph's Healthcare, McMaster University
The patient was admitted to a tertiary care hospital to undergoa
right upper-lobe wedge resection. Examination of the grossspecimen
through serial sections revealed a well-circumscribedcoin lesion
that measured 1.4 cm in diameter. Histopathologicexamination
revealed pulmonary infarction with central coagulativenecrosis
surrounded by a dense, fibrous capsule of mature granulationtissue.
A focus of foreign material was noted at the centreof the lesion.
The material was located entirely within a smallpulmonary artery. A
parasite resembling the immature form ofDirofilaria immitis
was identified on the histopathologic slides(Fig. 2).
The diagnosis of pulmonary dirofilariasis was confirmedby a
parasitologist at the US Centers for Disease Control andPrevention,
Figure 2. Photo: Images courtesy of the
Departments of Pathology and Molecular Medicine (HRLMP) and of
Diagnostic Imaging, St. Joseph's Healthcare, McMaster University
The results of 3 different tests of the patient's serum to detect
circulating D. immitis antigen (DiroCHEK and Witness Canine
Heartworm Antigen Test Kits, Synbiotics Corporation, San Diego,and
SNAP Heartworm Antigen Test, Idexx Laboratories, Westbrook,Me.) were
negative. The patient recovered completely withouttreatment.
The heartworm D. immitis is a ubiquitous parasite of dogs foundin many temperate parts of the world.1 It
is transmitted froman infected dog through the aspiration of
circulating microfilariae— the prelarval stage of the parasite — by
manyspecies of mosquitoes. The microfilariae mature in the mosquitoto third-stage larvae, which may then be transmitted by biteto
other dogs, domestic cats, wild canines and humans. The larvae
develop in subcutaneous tissues and migrate to the right sideof the
heart and pulmonary arteries, where they mature intoadult parasites.
In dogs, the adult worms produce microfilariaethat circulate in the
blood.2 In humans, D. immitis is
generallyunable to complete maturation, dies and forms an embolus inthe lungs. Thus, human disease is not associated with microfilaremiaor other clinical consequences. The negative results of the
dirofilarial antigen tests in our case are consistent with the
presence of only immature parasites. However, a low burden ofmature
parasites may produce similar results.
Human pulmonary dirofilariasis characteristically manifestsas a
solitary pulmonary nodule, which is easily confused withcancer.
About 120 cases were reported in the United States between1941 and
2000, and 60% of patients with pulmonary dirofilariasisare
asymptomatic.3 Human cases in Canada are
uncommon and areusually the result of travel to an endemic area.
Symptomaticpatients may experience pleuritic chest pain, cough or
hemoptysis.4Peripheral eosinophilia occurs in
less than 15% of cases. Chestradiographs usually show a
noncalcified, well-circumscribedperipheral coin lesion.5
The focal pulmonary infarct secondary to D. immitis infectiondiffers in shape from the classic thromboembolic infarct. The
latter is pyramidal, and the former is spherical, generally
attributed to the centrifugal diffusion of parasite antigenfrom the
degenerating worm.6 The histologic features
describedin our case are typical.5,7
Most symptomatic cases of human pulmonary dirofilariasis are
investigated by means of lung resection to rule out bronchogenic
carcinoma.2 Although the natural history of
pulmonary dirofilarialgranuloma is not well known, it is believed
that lesions ultimatelycalcify.4 No
specific therapy for pulmonary dirofilariasis isrequired.
Danny D. Lagrotteria Department of Medicine McMaster UniversityHamilton, Ont. Mark A. CrowtherChristine H. Lee Departmentof Molecular Medicine St. Joseph's Healthcare Hamilton, Ont.Andrew Peregrine Ontario Veterinary College Guelph, Ont.
Garcia LS, Bruckner DA. Diagnostic medical parasitology.
New York: Elsevier Science; 1988. p. 204-6.
Chesney TM, Martinez LC, Painter MW. Human pulmonary
dirofilarial granuloma. Ann Thorac Surg 1983;36(2):214-7.[Abstract]
Shah MK. Human pulmonary dirofilariasis: review of the
literature. South Med J 1999;92 (3):276-9.[Medline]
Ciferri F. Human pulmonary dirofilariasis in the United States:
a critical review. Am J Trop Med Hyg 1982;31:302-8.[Medline]
Kochar AS. Human pulmonary dirofilariasis: report of three cases
and brief review of the literature. Am J Clin Pathol 1985;84:19-23.[Medline]
Merrill JR, Otis J, Logan WD Jr, Davis MB. The dog heartworm (Dirofilaria
immitis) in man. An epidemic pending or in progress? JAMA
Neafie RC, Connor DH, Meyers WM: Dirofilariasis. In: Binford CH,
Connor DH, editors. Pathology of tropical and extraordinary diseases.
Washington: Armed Forces Institute of Pathology; 1976. p. 391-6.
My sister (8 years ago) had the same symptoms, heavy smoker,was feeling lousy all the time and they found something it looked as a small lung tumor.The doctors decide to open and get the mass out. They said they couldn't reach it.Opened again later and my sister (58) got an infection by stafilococcus aureous ,something it lives at the surgerie room.
She went into a comma for over two months and we lost her...I never knew till now about dog hearthworm till today.
Love to you.
Kim MK, Kim CH, Yeom BW, Park SH, Choi SY, Choi JS.
Department of Pathology, Guro Hospital, Korea University College of Medicine, Seoul, Korea.
Most of human dirofilariasis are pulmonary or subcutaneous infections, but there have been a few reports of human dirofilariasis in unusual sites, such as large vessels, mesentery, peritoneal cavity, and spermatic cord. We present the first case of human hepatic dirofilariasis, which was surgically diagnosed. A 39-year-old man without any evidence of systemic symptom was found incidentally to have a hepatic nodule during routine physical check-up. The histologic findings of the resected lesion showed a granulomatous lesion with central necrosis containing up to 35 transverse sections of a nematode, ranging 30-80 micro m in diameter. Thin (1.5-5 micro m) cuticle with transverse striations surrounded polymyarian and muscle bundles occupied a sixth of both sides of outer body cavity. Central portion of the body cavity was occupied with an intestine-like tubular structure and a larger reproductive tube. These microscopic findings were consistent with degenerated Dirofilaria immitis. Antibody test by enzyme-linked immunosorbent assay for patient serum reacted positively against adult D. immitis antigen.
Thanks you for your detail explanation. My question to you:
In an advanced heartworm case would it be safe to use a zapper? Is there any danger of the worms dying off too quickly. Until this problem, our 5-year old Lab has been extremely healthy and vigorous. I hope to hear from you soon.